Franks Aas Foundation

Deca Durabolin 16.5 ml (100 mg/ml)

Equipoise 33 ml (50 mg/ml) Testosterone Propionate 33 ml (100 mg/ml) Protest or Other 1 ml Benzyl Alcohol 0.5 ml Total = 84 ml * 2 ml Per Day For 42 Days

Parabolan 33 ml (22 76 mg Amps) Equipoise 33 ml (50 mg/ml) Testosterone Propionate 39 ml (100 mg/ml) Benzyl Alcohol 0.5 ml Total = 105.5 ml

Vial #1 allowed for 2 ml of Max Mix daily. Frank split this 1 ml per side for each body part worked that day. Testosterone really ramped up training intensity while aiding in post-work out rejuvenation. Equipoise worked well as a lean mass retention drug while adding to superior protein synthesis /sparing effects of Deca. I think Deca drew too much water when used by some individuals, even with a good estrogen suppression layer. Oust my opinion) So it was not my personal "best choice" during the last few weeks before a show.

Vial #2 allowed for 2.5 ml of Max Mix daily, obviously split 1.25 ml per side for each body part trained that day. Parabolan was an amazing AAS. It dramatically increased hardness, strength, and aided in fat burning. However, it was rough on the kidneys, so Frank needed to increase water intake until the last couple of days. (Masterone was successfully employed by some beasts to replace Parabolan as was Finabolan /trenbolone when timing factors are considered).

Example #1

The following example of layers Frank utilized (in a progressive manner) is pretty straight-forward. The AAS Foundation was set and estrogen control was layered in. Prolonged periods of AAS use significantly increased aldosterone production due to estrogenic activity and kidneys/adrenals stimulation. Control was a must or Frank would have appeared smooth and water logged. Nolvadex alone did not do it.

GH was layered into the AAS foundation beginning 8-12 weeks before his contest date in a daily protocol that slowly increased or ramped up in dosages. This aided in maintaining activity or counter acted the resulting up-regulation of somatostatin. The amino acid arginine was included to aid in somatostatin control. GH was removed 6-7 days prior to contest day as many athletes hold extra subcutaneous water during use. It takes 5-6 days for this to clear.

Due to calorie restriction and GH use, thyroid hormone activity decreased as a result or endogenous secretion/conversion down-regulation. For some beasts triacana was successfully utilized to restore and slightly elevate T-3 levels up to the last few days before a show.

The most common effective dosage was 1 mg/ 50 lbs of bodyweight daily divided into 4 equal dosages. More will not increase thyroid hormone activity. Triacana was also utilized on non-Cytomel days to assure adequate T-3 levels. Remember: triacana had a half-life of 6 hours.

Cytomel (T-3) was layered in beginning 6-8 weeks before a show. This was plenty for most beasts, unless they had gotten too fat during mass phases. The goal was to be lean and ripped, not to be hypothyroid for life or counter act all that anabolic/anti-catabolic chemistry. (Odds of this are very low but gambling is not a wise choice)

Thyroid hormone dosages had to be adjusted in respect to anabolic/anti-catabolic chemistry dosages. Not the other way around. This was how effective dosage levels were exceeded and long-term growth potential destroyed for all to many would-have-been-beasts. One step at a time!!

Cytomel was utilized in a 2 day on/2 day off protocol until the last 20-30 days before a show. So beginning 6 weeks out during a 12 week contest prep, dosages were: Week #7-8 50-100 mcg/d, #9-10 75-150 mcg/d, #1 1-12 100-200 mcg/d. Triacana was effectively utilized on non-Cytomel days and 100-200 mcg/d of Cytomel was utilized daily the last 20 days continuously.

Obviously the synergy between AAS and GH increased lean mass retention and/or growth (when thyroid hormone use was not excessive) and fat metabolization was significant. During the last 3 weeks before a show, Frank commonly utilized PGF-2 and / or IGF-1 to bring up weak body parts.

As mentioned prior, some body parts lost mass more quickly than others during calorie restricted periods. This would obviously potentially destroy symmetry. PGF-2 also aided in an over all hardening and fat loss effect. This was due to a synergistic response with T-3 and GH, but also through a different metabolic pathway. I have witnessed athletes adding 2-3 inches to their arms and/or calves during the last 3 weeks before a show.

Other muscle groups also had this response to site-specific PGF-2/IGF-1 administration. The average effective minimum IGF-1 dosage was 0.10 mcg/kg 3-5xd. The dosage for PGF-2 was 2 mg 3-5xd.

Beginning 8 weeks before a show, Frank utilized Clenbuterol and an Ephedrine / Caffeine stack in a 2 day each rotation. This also aided in T-4/T-3 conversion while providing some anti-catabolic qualities and increased thermalgenesis.

Beginning 8 weeks out, during a 12 week contest prep, an example of Frank's layer looked like this: Week #5-6 Clen. 80-100mcg/d-Eph 25 mg-Caff. 250 mg 3xd; #7-8 Clen. 100-120 mcg/d-Eph. 25 mg, Caff. 250 mg, #9-10 Clen. 120-140 mcg/d -Eph. 25 mg, Caff. 250 mg 3xd; #11-12 Clen. 140-160 mcg/d-Eph. 25 mg, Caff. 250 mg 3xd.

When Frank was not utilizing PGF-2, he increased Clenbuterol dosages an additional 20 mcg for each 2 week segment. Some beasts utilized 50-100 % higher dosages, but why would Frank have blown an effective threshold before necessary? Clenbuterol dosages were divided into 2-4 equal daily dosages due to that Frank and some other individuals experiencing some stomach discomfort with single dosage protocols.

Frank regularly monitored his body temperature to assure effective dosage and health. Over 101 degrees was too high, under 99.6 degrees was too low. (Elevated thyroid hormone levels also increased body temperature.)

"Just because it is done, does not mean it is right or best!" Coach

• Frank ceased CH on the last 6-7 days before a show. This aided in carb loading and water depletion both.

• During the last 1-2 days, Frank used 40 mg/d of Lasix to drop excess water weight.

• Frank had also successfully used 8-1 0 iu Humalog 2xd with 10 g creatine, 75 g dextrose, 25 g glutamine, 400 mg lipoic acid during the carb-loading period the last 2-3 days.

• Some beasts added PGF-2 week #10 2 mg 3xd, #11 2 mg 4xd, #12-2 mg 5xd.

Wfeek 1

Vial # 1-2 ml/d

GH 2 iu/2xd

Arimidex 1.5mg/d

Nolvadex 20mg/d

Week 2

Vial # 1-2 ml/d

GH 2 iu /2xd

Arimidex 1.5mg/d

Nolvadex 20mg/d

Week 3

Vial # 1-2 ml/d

GH 2 iu/2xd

Arimidex 1.5mg/d

Nolvadex 20mg/d

Week 4

Vial # 1-2 ml/d

GH 2 iu/2xd

Arimidex 1.5mg/d

Nolvadex 20mg/d

Week 5

Vial # 1-2 ml/d

GH 2 iu/3xd

Arimidex 1.5mg/d

Nolvadex 30mg/d

Week 6

Vial # 1-2 ml/d

GH 2 iu/3xd

Arimidex 1.5mg/d

Nolvadex 30mg/d

Week 7

Vial # 1-2 ml/d

GH 2 iu/3xd

Arimidex 1.5mg/d

Nolvadex 30mg/d

Week 8

Vial #2-2.5 ml/d

GH 2 iu/3xd

Arimidex 1.5mg/d

Nolvadex 30mg/d

Week 9

Vial #2-2.5 ml/d

GH 2 iu/4xd

Arimidex 1.5mg/d

Nolvadex 40mg/d

Week 10

Vial #2-2.5 ml/d

GH 2 iu/4xd

Arimidex 1.5mg/d

Nolvadex 40mg/d IGF-1 3xd

Week 11

Vial #2-2.5 ml/d

GH 2 iu/4xd

Arimidex 1.5mg/d

Nolvadex 40mg/d IGF-1 4xd

Week 1 2

Vial #2-2.5 ml/d

Arimidex 1.5mg/d

Nolvadex 40mg/d IGF-1 5xd

• Some did not respond to GH until daily dosages were at least 9iu

• Arimidex did possess "some" anti-catabolic /cortisol control qualities. However, Frank did best when he layered in a cortisol inhibitor the last 4 weeks before a show. 2 days on/2 days off was still best. He had employed three choices:

1. Cytadren-500 mg/d week #9, 750 mg/d week #10, 1000mg/d week #1 1-12.

2. Metryapone-500 mg/d week #9-12.

3. Trilostane -120 mg/d week #9, 180 mg/d week #10, 240 mg/d week #11-12.

• Remeron 1 5mg/d has been successfully used for this reason during the last 3 weeks.

It should be noted that prolonged periods of high dosage use of cortisol inhibitors (Except Remeron) has been reported to result in influencing ACTH production which in turn induces uncontrolled cortisol problems.

The use of different cortisol suppression drugs on alternating or consecutive schedules, would have been theoretically more beneficial over-all for delayed Action/Reaction Factors and negative feed-back loops.

An example utilized by a few beasts was that Metryapone inhibits 11-beta-hydroxylation in the adrenal cortex where as Trilostane inhibits the enzyme 3-beta-hydroxysteroid dehydrogenase delta 5,4 isomerase. This was two different metabolic pathways that accomplished the same goal of cortisol and/or estrogen inhibition.

Frank needed to regenerate proper HPTA function after the show since this protocol was lengthy. This meant hCg, Teslac, and Proviron or one of the methods explained else where in this book, or in "Chemical Muscle Enhancement".

Proviron has also been universally successfully utilized the last 3-8 week before a show to enhance over all hardness. This was also true of the liver hater, Halotestin, too. Male Mix also worked pretty well for post-cycle HPTA regeneration.

Frank layered in 6-10 iu 2xd of Humalog every other day during the last 4 weeks before a show only when he lacked over all size. Thankfully it was only once that this was deemed necessary. When a beast's size really sucked, he did the same during the first 4 weeks of a 1 2-week diet contest prep period as well. This allowed a 4 week off period between Insulin use.

Post competition, Frank was in a perfect state to begin an Absolute Anabolic Phase. (Gee, Ya Think?)

Note: Viagra and 3g of L-Arginine ingested an hour before each show resulted in a far better pump (not there) due to increased total body NO. This means improved vascularity and muscular harness with a fuller look.

Another approach to pre-contest fat loss utilized DNP (Dinitrophenol) in two different protocols. The first was simply a matter of ingesting 5 mg of DNP per KG of bodyweight daily for 7 days. Then begin an 8 week layered cycle. This cleared receptor-sites and made the body's musculature more sensitive to AAS and other protocols.

It also caused a 4-8 LB fat loss while preventing the need for an entire 12-16 week period of AAS use. This was possible only when a beast did not have too much skin to tighten up from getting sloppy during mass gaining phases...

The second method involved the use of DNP on one or two weekly non-training days (with aerobics) only. This DNP/non-training day(s) was best at the beginning of each week for 8 consecutive weeks. T-3 such as Cytomel was usually layered in as well and the total cycle length ran 60 days. This allowed for greater receptor sensitivity each week. The AAS Foundation was either a Max Mix or as listed, assuming a lean mass weight of 271 lbs.

GH use was 2 iu 2xd day #1-20, 2 iu 3xd day #21-40, 2 iu 4xd day # 41-57 and 2 iu 2xd on day #5860.

Cytomel (T-3) use was 50-100 mcg on days #4-30, 75-1 50 mcg on days #31-45, 100-200 mcg on days #46-60 (on listed days) and Triacana on non-T-3 days.

Estrogen Control began on day # 1 and continued for atleast 1 0 days after day # 60 to limit negative post-cycle feed-back loops.

Cortisol Suppression was sometimes layered in beginning day # 30 and ran a total of 30 days. 2 on/2 off. This was long enough to significantly reduce an overwhelming cortisol level post-cycle as well.

IGF-l/PGF-2 was sometimes layered in during the last 21 days on non-DNP days.

Insulin (Fast acting) was an additional layer sometimes employed on DNP days only equal to carb intake post work-out and during the 2-3 days of carb-loading.

Thermalgenics like E/C/A helped some beasts reduce carb craving on DNP days. Glutamine was excellent for this purpose also.

DAY

DRUGS

DAY

DRUGS

1.

DNP/Sus-2 50/Deca 250 mg/GH

31.

GH/T-3

2.

GH

32.

GH/T-3

3.

GH

33.

Sus-250/Primo D. 200 mg/GH/T-3

4.

GH/T-3

34.

T-3/Win. D. 50mg/GH

5.

GH

35.

T-3/Win. D. 50mg/GH

6.

GH

36.

DNP/Win. D.50 mg/GH

7.

GH

37.

Sus-250/Primo D. 200 mg/GH/T-3

8.

DNP/GH/T-3

38.

T-3/Win.D. 50 mg/GH

9.

Sus.-250/Deca 250 mg/GH/T-3

39.

Win. D 50mg/GH/T-3

10.

GH

40.

Win. D 50mg/GH/T-3

1 1.

GH

41.

Sus-250/Primo D. 200 mg/GH/T-3

12.

GH/T-3

42.

T-3/Win. D. 50mg/GH

13.

Sus.-250/Deca 250 mq/GH/T-3

43.

DNP/Win. D 50 ma/CH

14.

GH

44.

T-3/Win. D. 50mg/GH

15.

DNP/GH

45.

T-3/Test. P. 150 mg Primo D 200 ma/GH

16.

GH/T-3

46.

Win. D 50mg/CH/T-3

17.

Sus.-250/Deca 250 mg/GH/T-3

47.

Test. P. 1 50mg Win. D. 50 mg/GH/T-3

18.

GH

48.

T-3/Win. D 50 mg/GH

19.

CH

49.

T-3/Test. P. 150 mg Primo D 200 mq/GH

20.

GH/T-3

50.

DNP/Win. D 50 mg/GH

21.

Sus.-250/Deca 250 mg/GH/T-3

51.

T-3/Test. P. 1 50 mg Win. D. 50mg/GH

22.

DNP/GH

52.

Win. D 50 mg/GH/T-3

23.

GH/T-3

53.

T-3/Test. P. 1 50 mg Win. D. 50mg/GH

24.

GH/T-3

54.

Win. D 50 mg/GH/T-3

25.

Sus.-250/Deca 250 mg/GH

55.

T-3/Test. P. 200mg Win. D. 50 mg/GH

26.

GH

56.

T-3/Win. D 50 mg/GH

27.

GH/T-3

57.

Test. P. 200 mg Primo D. 200mq/GH/T-3

28.

GH/T-3

58.

Win. D. 50 mg/GH/T-3

29.

DNP/Sus-2 50/Deca 250 mg/GH

59.

T-3/Test. P. 200 mg Win. D. 50 mq/GH

30.

GH/T-3

60.

T-3/Win. D 50 mg/GH

• Triacana was sometimes added on non-T-3 days

I realize some may feel that the dosages listed in any example thus far were low. There were individuals who I knew that introduced as much as 3000 mg (even 6000 mg) of AAS alone weekly. Just because it was done, did not make it right... or necessary.

Personally, I did not believe that a weekly AAS actual "plasma threshold" should have ever exceeded 1500 mg. (Remember: This issue relates to "plasma threshold" and not to the ester weight adjoined to the active AAS itself.) If it had become necessary to do so to make gains, then it would have been time to focus upon clearing/up-regulating androgen receptor-sites and re-evaluate Action/Reaction Factors.

In most cases, those individuals who failed to realize results at 1 500 mg weekly plasma levels were those whose phases were fighting the body's adaptive response mechanisms. Not properly utilizing these adaptive responses for maximal progress always resulted in failure eventually.

Another example of a plan to fail was the abuse of cortisol inhibitors such as Cytadren. 2500 mg/d had become common during contest prep for less knowledgeable individuals. What on earth made someone believe they needed a dosage above that of a Cushings Syndrome patient?

2000 mg/d totally inhibits hormone biosynthesis for Cushings victims and they are producing many times more cortisol than the hardest training mega dosed bodybuilder ever was. To add to this I lost a dear and close friend not long ago. He had been in a minor car crash. The poor doctors could not stop the bleeding due to my friends secretive over-use of Cytadren.

Take a good look at some of the Masters Olympia lads. Vince Taylor was well past 40 years old and he looked better than ever. Vince had been a top competitor for 20 some odd years and the man was healthy. Do you honestly believe he utilized life long mega dose protocols? He would have been dead or totally burned out if so. I hope he, and several other greats, continue as they have. They will actually get better still. It's a fact!

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