How To Cure Peripheral Neuropathy Naturally

Peripheral Neuropathy Solution By Dr. Randall Labrum

Neuropathy Solution is considered as a self- treatment program that supplies people with a proven, simple solution for peripheral neuropathy. Randall Labrum, the author of this program claims that his treatment works successfully for most cases without fail, no matter your peripheral neuropathy results from chemotherapy, diabetes, hypertensions, or aging process. Neuropathy Solution is vastly more efficient than drugs as it targets and corrects the underlying cause of the Neuropathy with a series of techniques that can work in natural harmony with your bodys healing abilities and restorative functions. You can effectively heal your neuropathy and achieve optimal health. With the Neuropathy Solution Program, you do not need to squander more money and time on surgery and drugs, as well as other methods that do not function. Many people have benefited from this program. If you give it a try, you too can gain those benefits. Read more here...

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Helping Diabetic Neuropathy

People with diabetes sometimes get a painful nerve condition called diabetic neuropathy. We still don't know exactly what causes the problem, but lipoic acid can often help. The doses needed are fairly high. In Europe, where doctors can prescribe lipoic acid, the usual dose is 600 to 1,200 mg a day. It has been widely used in Germany for over 25 years for treating diabetic neuropathy.

Brief Lesson In Anatomy And Physiology

CENTRAL NERVOUS SYSTEM (CNS) The spinal cord and brain. The cns functions in conjunction with the peripheral nervous system, which consists of the ganglia and nerves that reside outside of the brain and spinal cord. The nervous system appears like thousands ot little wires that function as transmitters, receivers, and interpreters ot data from all parts of the body. It is responsible for stimulating the muscles of your body to contract. It is of vital importance to both the aspiring and competitive bodybuilder as, without nerves, we'd be immobilized because our muscles wouldn't contract.

Gene Therapy for Laminin a2Deficient Congenital Muscular Dystrophy

Laminin a2-deficient congenital muscular dystrophy (MDC1A) is a very severe, autosomal recessive muscle wasting disease that often leads to death in early childhood (Allamand and Guicheney 2002 Emery 2002). It is caused by mutations in LAMA2, the gene encoding laminin a2, which assembles with the p1 and the g1 chain to laminin-2, the main laminin isoform present in the basement membrane of muscle fibers and peripheral nerves (Meinen and Ruegg 2006). Basement membranes are highly structured sheets of extracellular matrix molecules that laminate a variety of cells including myofibers. The major role of laminin-2 in the muscle is to interconnect the myofiber extracellular basal lamina with the plasma membrane. In laminin-a2 deficient individuals, the basal lamina is corrupted and the trans-membrane cytoskeletal structure is lost. As a consequence, muscle fibers lose their stability and degenerate.

Mutations in nuclear genes affecting mtDNA maintenance

The first description of a disease with autosomal-dominant inheritance and multiple mtDNA deletions goes back to 1989 (Zeviani et al., 1989). Multiple mtDNA deletions have been associated with several clinical manifestations, which in most cases present after the first decade of life. The most common, albeit not constant, symptom is progressive external ophthalmoplegia (PEO) with the variable addition of exercise intolerance, recurrent myoglobinuria, ataxia, parkinsonism, major depression, peripheral neuropathy, hypogonadism and cardiomyopathy (Zeviani et al., 1990 Cormier et al., 1991 Ohno et al., 1991 Servidei et al., 1991 Haltia et al., 1992 Suomalainen et al., 1992a Prelle et al., 1993 Checcarelli et al., 1994 Kawashima et al., 1994 Takei et al., 1995 Ville-Ferlin et al., 1995 Bohlega et al., 1996 Campos et al., 1996a Chalmers et al., 1996 Fabrizi et al., 1996 Melberg et al., 1996a Suomalainen et al., 1997 Carrozzo et al., 1998 Federico et al., 1998 Melberg et al., 1998 Nishizuka...

Biomechanics of the Craniosacral System

Falx And Ligaments Within The Skull

The spinal dura mater encloses the spinal cord and follows the peripheral nerves up to the intervertebral foramen, where it passes into the outer cover of the nerves. In the intervertebral foramen it is also affixed to the bone. Furthermore, there are relatively loose attachments at the vertebral bodies via the denticulate ligaments.

Stimulation Tests Rationale

Peripheral nerve, spinal, or cortical stimulation, either by implanted electrodes (for peripheral nerves) or by externally applied electric or magnetic fields, elicit relatively synchronized activation of motor units at reproducible and predictable levels. The resulting compound action potentials and subsequent muscle contraction allow for measurement of the efficiency of neural and neuromuscular transmission. The muscle responses to stimulation are discussed in Phrenic Nerve Stimulation in Section 2 of this Statement.

Autosomal dominant form

A-type lamin is essential for maintaining the architecture and structural integrity of the nuclear envelope, and the observation that loss of A-type lamins causes both mechanical weakness and defects in mechanical-stress-dependent gene expression in vivo fed the mechanotransduction hypothesis. However, it does not explain the pathology of other tissues caused by LMNA mutations such as abnormal fat distribution in lipodystrophy and peripheral neuropathy in Charcot-Marie-Tooth disease.

Nervous system and ocular manifestations

Cushing Syndrome Addison Disease

The central nervous system (CNS), peripheral nervous system (PNS) and skeletal muscle are affected in the majority of mitochondrial disorders. The signs and symptoms from the nervous system may include mental retardation, autistic features, mental depression, apnea attacks, myoclonic seizures, epilepsy, ataxia, migrainelike headache, stroke-like episodes, sensorineural hearing loss, reduced vision and blindness, hemianopsia, peripheral neuropathy, muscle weakness and fatigue, muscle hypotonia and hypertonia, ptosis and ophthalmo-paresis, autonomic disturbances and others. The CNS Peripheral neuropathy is commonly encountered in mitochondrial diseases. Axonal degeneration is the most frequent pathological change but primary myelin damage may occur (Schroder, 1993 Bouillot et al., 2002). Motor neuron disease and spinal muscular atrophy have been described in association with OXPHOS diseases (Comi et al., 1998 Kirches et al., 1999 Berger et al., 2003 Tarnopolsky et al., 2004).

The Importance Of Carbohydrates

Carbohydrates, next to water, are by far the most important nutritional element anybody, not just bodybuilders, could consume. The most important reason is that our nervous systems, our brains, our spinal cords, and our peripheral nerves derive 99.9 percent of their nutrition from the one thing that we've been taught is the worst thing we can eat sugar. In fact, your brain derives 99 percent of its nutrition from sugar. Carbohydrates are the most important thing you can eat because they are the body's source of glucose, the sugar that is its basic fuel.

Autosomaldominantrecessive PEO with multiple mtDNA deletions

In ad ar PEO, muscle weakness and exercise intolerance are the most common symptoms. Additional manifestations, which sometimes may be the presenting symptoms, include peripheral neuropathy, ataxia, tremor, parkinsonism, mental depression, cataracts, pigmentary retinopathy, dysphagia, hypoacusia, episodic rhabdomy-olysis and hypogonadism (Zeviani et al., 1990 Servidei et al., 1991 Kaukonen et al., 1996 Melberg et al., 1996a 1996b Suomalainen et al., 1997 Melberg et al., 1998 Kaukonen et al., 1999). The MNGIE syndrome includes gastrointestinal manifestations, cachexia, leuko-encephalopathy, peripheral neuropathy and mitochondrial myopathy (Nishino et al., 2001).

The Osteopathy of Dr Still

Still describes the cerebrospinal fluid (CSF) as perhaps the most important known element ( the highest known element ) of the whole organism. In its constituents, it resembles the blood and lymph serum. It is connected to both of these fluids with the blood via the plexus chorioidei and with the lymph via the peripheral nerves in the interstitial space. In addition to its supportive and nutritive functions for the central nervous system, Still, and especially his student Sutherland, accorded the CSF another special significance together with the fluid, the breath of life infiltrates all body cells.54'14a 142-143

Cardiac Muscle The Autonomic Nervous System

Somatic nervous system, the motor commands exiting from the central nervous system go directly to the target cells, while in the autonomic nervous system, the motor commands from the central nervous system are relayed via an additional synaptic connection in the peripheral nervous system.

Clinical aspects

Recently mutations in dynamin 2 (DNM2), have been identified in a subset of patients with autosomal dominant centronuclear myopathy (Bitoun et al., 2005). Dynamins are a family of GTPases involved in vesicle trafficking. DNM2 has also been shown to be mutated in autosomal dominant intermediate Charcot-Marie-Tooth disease, associated in some cases with neutropenia, suggesting that DNM2 plays a role in maturation of neutrophils as well as peripheral nerves and muscle (Zuchner et al., 2005). Arthrogryposis multiplex congenita (AMC) is a syndrome characterized clinically by congenital fixation of multiple joints. In distal arthrogryposis (DA) infants are born with congenital contractures of distal limbs (Sung et al., 2003a). Arthrogryposis multiplex congenita and DA can be secondary to a wide range of disorders, including disorders of the neuraxis such as lissencephaly, meningocele, sacral agenesis, anterior horn cell disorders such as spinal muscular atrophy, peripheral nerve...

Magnetic Stimulation

Schematic representation of the principle of magnetic stimulation. The electric current produced by the charging circuit is stored in capacitors. Switching on the trigger circuit results in a sudden pulse of electric current in the coil of wire held in contact with the patient. The intensity of this pulse of current can be modulated (from 0 to 100 of maximal intensity). A pulsed magnetic field orthogonal to the current flowing through the coil is produced. This magnetic field is able to penetrate body tissues such as skin or bone with little attenuation. It in turn induces secondary electric currents of low intensity in deep structures. If such a structure, for example, the cerebral cortex or the trunk of a peripheral nerve, lies at a tangent to the secondary electric currents, it is depolarized at a point that depends on the local geometry and on the intensity of the current. The intensity of the magnetic field induced within the tissues decreases with distance. The...


Dichloroacetate, an inhibitor of pyruvate dehydro-genase kinase, decreases lactate concentration effectively in most patients with lactic acidosis (Stacpoole et al., 1997). However, there is little evidence of associated clinical benefit, except in a few patients with MELAS and LS (Saijo et al., 1991 Taivassalo et al., 1996 Kuroda et al., 1997 Takanashi et al., 1997 Saitoh et al., 1998 Fujii et al., 2002 Mori et al., 2004). A common side effect of dichloroacetate treatment is that it may aggravate or cause a peripheral neuropathy (Stacpoole et al., 1997 Spruijt et al., 2001 Kaufmann et al., 2006). The neuropathy is considered to be a result of secondary thiamine deficiency caused by an increased demand for this vitamin associated with dichloroacetate therapy. Co-administration of thiamine may therefore decrease the risk for dichloroacetate-associated neuropathy (Stacpoole et al., 1997 Fujii et al., 2002). A mild liver dysfunction has also been noted in patients with MELAS on long-term...

MELAS syndrome

Brain Mitochondrial Mutation A3243g

Myoclonus is usually stimulus sensitive. Seizures are tonic-clonic, often with photosensitivity. Other common features are short stature, sensorineural hearing loss, optic atrophy, dementia, peripheral neuropathy and spastic paraparesis. In a review of 62 patients with MERRF syndrome and the A8344G mutation, one-third had cardio-myopathy presenting either in adolescence or adulthood (Hirano and DiMauro, 1996). A few patients have multiple neck and truncal lipomas. Clinical features of affected relatives vary in pattern and severity of symptoms. Age of onset differs among family members. Many relatives are asymptomatic with low levels of mutated mtDNA (Larsson et al., 1992).

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