Dirtiest Diet Award

1. DNP 6 MG/KG-CYTADREN 250 MG 2XD

2. DNP 6 MG/KG-CYTADREN 250 MG 2XD

3. DNP 6 MG/KG-NOLVADEX 20 MG AM/PM -ARIMIDEX 0.5 MG 3XD

4. DNP 6 MG/KG-NOLVADEX 20 MG AM/PM -ARIMIDEX 0.5 MG 3XD

5. DNP 6 MG/KG-CYTADREN 250 MG 2XD -PRIMOBOLAN DEPOT 100 MG -TEST. PROP. 300 MG

6. TEST. PROP.-300 MG-CYTADREN 250 MG 250 MG 2XD-PRIMO DEPOT 100 MG

7. TEST. PROP. 300 MG-NOLVADEX 20 MG AM/PM -ARIMIDEX 0.5 MG 4XD-PRIMO DEPOT 100 MG

8. TEST. PROP. 300MG-NOLVADEX 20 MG AM/PM-ARIMIDEX 0.5 MG 4 XD-PRIMO DEPOT 100 MG

9. TEST. PROP. 300 MG-CYTADREN 250 MG 3XD-PRIMO DEPOT

10. CYTADREN 250 MG 3XD

11. DNP 6 MG/KG-NOLVADEX 20 MG AM/30 MG PM-ARIMIDEX 0.5 MG 4XD

12. DNP 6 MG/KG-NOLVADEX 20 MG AM/30 MG PM-ARIMIDEX 0.5 MG 4 XD

13. DNP 6 MG/KG-CYTADREN 250 MG 3XD

14. DNP 6 MG/KG-CYTADREN 250 MG 3XD

15. DNP 6 MG/KG -NOLVADEX 20 MG AM / 30 MG PM-ARIMIDEX 0.5 MG 5XD-PARABOLAN 76 MG

16. GH 2 I.U. 3XD-NOLVADEX 20 MG AM/30 MG PM-ARIMIDEX 0.5 MG 5XD-PARABOLAN 76 MG -CYTOMEL 50 MCG AM

17. GH 2 I.U. 3XD-CYTADREN 250 MG 4XD-CYTOMEL 50 MCG AM-TEST. PROP. 200 MG-PARABOLAN 76 MG

18. GH 2 I.U. 3XD-CYTADREN 250 MG-4XD-TEST. PROP. 200 MG-PARABOLAN 76 MG

19. GH 2 I.U. 3XD-CYTOMEL 50 MCG AM-NOLVADEX 20 M.G. A.M./40 M.G. P.M. ARIMIDEX 0.5 M.G. 5XD-TEST. PROP. 200 M.G.-SPIRONOTHIAZID 25 M.G.

20. G.H. 2 I.U. 3XD-CYTOMEL 50 M.C.G. A.M.-NOLVADEX 30 M.G. A.M./30 M.G. P.M.ARIMIDEX 0.5 M.G. 5XD-SPIRONOTHIAZID 50 M.G.

21.CYTADREN 250 M.G. 4XD-NOLVADEX 30 M.G. A.M./ 30 M.G. P.M. ARIMIDEX 0.5 M.G. 5XD-SPIRONOTHIAZID 50 M.G

What was that, huh? There was a bodybuilder who stated with tears, " I have 22 days before a show. If I don't compete, I lose my contract." He was 250 pounds at 9 % bodyfat which is 22.5 LBS of fat total. To reach 4 %, he needed to drop about 12.5 LBS of fat and a great deal of water. At least he was not 15% bodyfat. He followed this protocol and actually looked great, but he won the dirtiest diet award.

The strategy he utilized was a high estrogen suppression/high cortisol suppression protocol. He had an added problem of being one of those who encounter the paradox listed in "Nolvadex". His body reacted by over producing DHEA. The Cytadren eliminated that problem quite well.

By utilizing brief high dosages of DNP and successfully timing Primobolan Depot and Parabolan, he was able to retain almost all of his lean mass. GH/Cytomel aided in kicking up metabolism and increased protein absorption. He ate 500 grams of protein daily and broccoli 6 times daily adding flax and olive oil. He also drank 30 ML of glycerine in 16 oz of water daily beginning day #1, then twice daily (AM & PM) beginning day 6. 20 grams of glutamine was ingested daily, divided into 5 separate dosages with 2 grams of vitamin c 3 times daily. On non-DNP days, he utilized 50 MG of ephedrine and 325 MG of caffeine twice daily. He carb-depleted on day #17 &18 and carb-loaded on day #19-21.

Since his body was not producing much Aldosterone (the hormone that causes most water retention) he was able to dump most of his remaining water using Spironothiazid for the last 3 days which of course also suppresses Aldosterone. He looked fairly dry and tight at 219 pounds on contest day with full muscle bellies due to Testosterone Propionate's reputed unique carb loading qualities and Parabolan's high androgenic profile. A few had utilized this protocol also but added Winstrol Depot on DNP days. The lad was a furnace the entire 3 weeks but swears he felt good on contest day. What can I say except sometime odd things worked.

There are several protocols which were very effective in a 28 day period. Utilized properly they were notably healthier than any 12 week AAS protocol and very effective. The only problem I had encountered was seriously bloated bodybuilders. 28 days is not enough time for skin to tighten up in some cases. The crying competitor is a good example of action/reaction and working with, instead of against, the body. He had spent months gaining quality mass and the body naturally fought back attempting homeostasis by way of weight loss. He simply helped it and switched priority toward weight loss from liposytes (fat cells) instead of protein based tissues. Since he also ingested Omega 3 and 6 fatty acids (flax and olive oils) his body did not fight him much, nor did he compromise his health as much as most do with crazy fat less diets. But I must add as the supreme skeptic that I wonder about some long term side effects he may experience. He was also smart enough to keep water intake high until day 18 through continuous sipping all day long. As we know by now, cortisol, estrogens, and aldosterone were the big enemies of cuts and separation.

ANDROGENS AND LIBIDO

Part of the attraction of anabolic/androgenic steroid and pro hormone use was a distinct increase in sex drive and sexual pleasure. Those readers who have utilized androgens know the joy of being seriously horny and the elevated sensitivity of sexual arousal. The down side was post-cycle libido crash many experienced for 2-12 weeks following initial androgen discontinuance. How much was libido elevated during androgenic usage? Watch a woman who is employing androgen cycles ride an exercise bike sometime. In some cases it is a better voyeuristic experience than the strip-tease scene in True Lies.

Let's look at sexuality from a biological stand point first. (Sorry, pictures just do not seem appropriate). There are a multitude of receptors in the brain that constantly monitor internal biochemistry. In some cases, specific substances activate receptors that alter behavior and sensations. Androgens activate, or stimulate, receptors in the preoptic aspects of the hypothalamus. These aspects respond by increasing the sensitivity of the pudendal nerve that provides tactile data from the pubic area. These sensations make us more easily stimulated (horny). Heightened sensitivity is not the whole equation however. Though a strong wind was enough in high school to be cause for a large book held low. Cortical activity is another piece of the puzzle. Neurotransmitters are chemicals our neurons utilize to communicate with each other and with different areas of our bodies. Dopamine is a neurotransmitter which is released by neurons in the medial preoptic area of the hypothalamus in response to male sexual activity, such as sexual arousal. In turn, an erection results when dopamine activates D-1 receptor sites within the brain. This is due to moderate levels of dopamine. When dopamine levels increase above moderate levels, the D-2 receptor sites are activated which causes male ejaculation.

When exogenous forms of androgens are introduced into the body, various peptide amino acid levels increase. These amino acids can act as neuromodulators which inhibit or prolong the effects of neurotransmitters. Including the role of dopamine in sex. This means our cortical androgenic receptors (and obviously related sexual response mechanisms) send and receive signals, or impulses, to various sites which enhance sensory and behavioral processing activity that maintains our ability to have sex as well as sex drive. When supraphysiological levels of androgens are introduced from outside sources, there is an over abundance of receptor site stimulation in the hypothalamus and a subsequent increased production of neuromodulating peptides. This in short, makes people very horny during the first 3-8 weeks of an androgenic cycle. After a period of time...

these receptors-sites and sexual response areas adjust to these heightened levels and libido returns to normal. Unfortunately post-cycle androgen levels are even lower than pre-cycle levels due to androgen induced suppression of the HPTA. This results in desensitized receptor-sites and sexual tissue. This is partially due to heightened stimulation thresholds in the pudendal nerve (which would decrease sexual sensitivity). Of course, the post-cycle decrease in dopamine release and uptake can limit our ability and desire to have sex as well. All of this plus psychological factors can, in some cases, lead to a lack of sexual interests, and even erectile disjunction.

Though I have noted no permanent post-cycle dysfunction in any one, as of yet, it is possible. This is more due to psychological factors than with biological. In most cases, the use of HCG and/or Clomid quickly returned normal sexual interest, but I am sure some have sought psychological help as well.

Some interesting methods (which no doubt will be cause for nasty letters from OB GYNS) that may quickly solve the problem are simple. L-Dopa is a precursor for dopamine and is available in health food shops selling herbal remedies. Another playtime idea involved rubbing HPC cyclodextrin forms of 4-androstenediol into the clitoris, penis, and testes areas. Liposomal or topicals worked also, but some reported that they would have been wiser to watch out for those that include alcohol in their formulas. GH also aids in receptor regeneration but there is little need in most cases. Not a suggestion of course, just points of interest.

COMING OFF LONG AAS CYCLES

Many top competitors in bodybuilding and power lifting stayed on AAS cycles for years. This was due to many factors (none of which were health). For bodybuilders, it was a matter of being in top shape so as to make a living from guest appearances, and the almost year-long competition circuits now common. For all, the loss of performance and muscle mass created a real concern.

As explained in the beginning of this book, anabolic/androgenic steroids (AAS) have strong anti-catabolic effects as well. While an athlete was administering exogenous androgens at a much higher level than the HPTA naturally produces, the body had been trying to re-establish homeostasis (an equilibrium or balance between anabolism and catabolism) by over producing cortisol from the adrenal glands. As you know, cortisol is a very catabolic (tissue wasting) hormone. This was okay during high androgen levels from exogenous sources (occurring outside the body) because the AAS molecules were blocking the cortisol receptors. Thus preventing the cortisol from entering and down-regulating muscle mass build- up. Once the exogenous androgen supply was gone (evil music please...) the high level of cortisol raped and pillaged that newly built muscle mass. Second: As you realize, all anabolic /androgenic steroids decrease HPTA function to some extent. This means natural testosterone production was either lower or non existent. So the body was not able to restore natural anabolism/catabolism homeostasis with much help from the Leydig's cells in the testes. Third; due to the aromatization of most steroids, estrogen levels were high and estrogen sometimes became the dominant hormone. You will bench like a sissy too!

Before males put on a dress and females refuse to come off steroids, please realize much of this was reported to be avoided. Note: I said "much", and not "all". The fall from high androgenics was worse than the milder high anabolic. This was mostly due to longer AAS use periods and aromatization issues.

The following was theoretical information I once offered during an interview that was in reply to the question "What if someone could not handle the psychological issues relating to coming off steroids and the eminent shrinking?"

Start by setting a stop date. Set your mental state as "going into the next phase" not the end of an era. Do this right and you will keep much of your AAS aided mass gains while losing some bodyfat.

Chemically Engineered

Chemically Engineered

Push Beyond Your Genetic Potential Using Steroids To Build Massive Muscle. If you grew up as a skinny geek like I did then you understand why some people decide to use steroids to push beyond the limits that nature left them with.

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